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MYC Antibody (OAEE01021)
เพิ่มเมื่อ: 2017-01-20 11:01:39.0
รายละเอียด:
AVV-OAEE01021
AVV-OAEE01021
- TypeMonoclonal Antibody
- SpecificityThe antibody 9E10 may be used to detect the c-Myc tag.
The c-myc gene (8q24 on human chromosome) is the cellular homologue of the v-myc gene originally isolated from an avian myelocytomatosis virus. The c-Myc protein is a transcription factor (nuclear localization). c-Myc is commonly activated in a variety of tumor cells and plays an important role in cellular proliferation, differentiation, apoptosis and cell cycle progression. The phosphorylation of c-Myc has been investigated and previous studies have suggested a functional association between phosphorylation at Thr58/Ser62 by glycogen synthase kinase 3, cyclin-dependent kinase, ERK2 and C-Jun N-terminal Kinase (JNK) in cell proliferation and cell cycle regulation. In normal cells the expression of c-Myc is tightly regulated but in human cancers c-Myc is frequently deregulated. c-Myc is also essential for tumor cell development in vasculogenesis and angiogenesis that distribute blood throughout the cells. - Host
- ApplicationFC, IP, WB, IHC-P
- CompanyAVIVA
- Catalog No.AVV-OAEE01021
- Product nameMYC Antibody (OAEE01021)
- IsotypeIgG1
- Size0.1 mg
- LinkDataSheethttp://www.avivasysbio.com/sd/tds/html_datasheet.php?sku=OAEE01021
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Catalog No.
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AVV-OAEE01021
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Product Name
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MYC Antibody (OAEE01021)
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Company
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AVIVA
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Type
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Monoclonal Antibody
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Size
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0.1 mg
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Isotype
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IgG1
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Application
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FC, IP, WB, IHC-P
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Specificity
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The antibody 9E10 may be used to detect the c-Myc tag.
The c-myc gene (8q24 on human chromosome) is the cellular homologue of the v-myc gene originally isolated from an avian myelocytomatosis virus. The c-Myc protein is a transcription factor (nuclear localization). c-Myc is commonly activated in a variety of tumor cells and plays an important role in cellular proliferation, differentiation, apoptosis and cell cycle progression. The phosphorylation of c-Myc has been investigated and previous studies have suggested a functional association between phosphorylation at Thr58/Ser62 by glycogen synthase kinase 3, cyclin-dependent kinase, ERK2 and C-Jun N-terminal Kinase (JNK) in cell proliferation and cell cycle regulation. In normal cells the expression of c-Myc is tightly regulated but in human cancers c-Myc is frequently deregulated. c-Myc is also essential for tumor cell development in vasculogenesis and angiogenesis that distribute blood throughout the cells. |
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Product URL
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